Dystonia is a challenging neurological disorder characterized by involuntary muscle contractions causing twisting and repetitive movements or abnormal postures. While there is currently no cure for dystonia, various treatment options are available to reduce its symptoms and improve quality of life.

Despite the incomplete recapitulation of human MSA pathology [a hundred seventy five, 177] preclinical reports have taught us crucial classes around the pathophysiological cascade of MSA and secondary variations attributable to aberrant ?-synuclein aggregation [178]. These products opened an avenue

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